
Brain Related Drug Development
What is PRV-002?
PRV-002 is a new chemical entity (NCE) with proven in-vivo efficacy in animal concussion models through gene amplification of anti-inflammatories, antioxidants, and anti-edematous channels.
Safety margin > 200 fold in animal toxicology studies
The IP covers composition of matter, synthetic routes, and methods of use
Mouth to Nose (breath propulsion) delivery system
Nano-particle spray-dried formulation drives drug into the brain in minutes
How does PRV-002 work?
PRV-002 induces an intracellular steroid receptor found in brain cells. The stimulation of PRV-002 receptoractivates multiple gene response elements promoting transcription of anti-inflammatories, anti-oxidants, and efflux channels that remove excess fluid and debris. Since PRV-002 is not a GABAergic compound, it avoids drug-induced fatigue and any cognitive decline associated with prolonged use. PRV-002 weakly activates the progesterone receptor, avoiding side effects such as clotting or infertility.
In development, not commercially available.

What is PRV-001?
PRV-001 is a First-in-Class Neurosteroid for the treatment of Niemann Pick Type-C Disease.
The IP covers methods of preparation and methods of use
There are very few competing products currently in human trials
Animal and cell culture models of neuronal injury show positive preclinical effects
There has been no drug-related toxicity observed
The intranasal formulation is complete
How does PRV-001 work?
PRV-001 binds to an intracellular steroid receptor. The PRV-001 receptor is an intracellular receptor found in neurons, glia and the endothelium of the blood brain-barrier. The induction of the PRV-001 receptor activates the gene response element leading to the transcription of P-Glycoprotein (PGP), which pumps toxins, cholesterol, and lipid debris, as well as excess fluid, out of the brain cells and brain circulation. Since PRV-001 is not a GABAergic compound, it causes no fatigue. Furthermore, PRV-001 promotes neuronal synatopgenesis in neuronal cell culture models.
PRV-001 Toxicology
Acute and sub-acute dose ranging studies were executed in rats – intranasal and intravenous. No drug-related effects were noted in hematology, blood chemistry, BW, FC, or histopathology.
What is Niemann Pick Type- C Disease?
Nieman Pick Type-C Disease is a rare brain disorder in children. It is considered an “orphan indication” by the FDA and therefore has special considerations in the FDA approval process.
Niemann-Pick Type C disease (NPC) is an autosomal recessive, lysosomal storage disorder characterized by accumulation of cholesterol and gangliosides in the brain and liver.
NPC is a rare (estimated U.S. prevalence of 1:150,000) neurodegenerative disorder. Prevalence is 5X higher with Arabian decent.
Children with NPC demonstrate neurological dysfunction with cerebellar ataxia, dysarthria, seizures, visual palsy, motor impairment, dysphagia, psychotic episodes, and progressive dementia.
There is no effective treatment for NPC, and it is a lethal disorder.